copd. Views: 481, By: DrDwayne The good news is COPD is often preventable and . Now customize the name of a clipboard to store your clips. Center of disease control and prevention. -Partner with the Center for Education, Simulation, and Innovation to develop patient scenarios that foster the development of critical thinking skills. 21 slides. experiencing shortness of breath at rest or with minimal activity, such as walking from one room to another. Activate your 30 day free trialto continue reading. Comment The A-a gradient calculation for patient 2 is as follows: PAO2 = 150 (1.25 x PCO2) PAO2 = 150 (1.25 x 50) PAO2 = 150 63 PAO2 = 87 Therefore, A-a = 87 50 =37 (an abnormally increased gradient). The inclusion criterion for nurses was employment in home nursing care for at least 6 months. You can read more about the PCG tracks for patients and caregivers and for health care professionals. COPD - Physical Findings. Quieres ms informacin?Llame al 866.731.COPD (2673) y presione 9 para hablar en espaol con un paciente o cuidador. Background: If current trends in the management of COPD do not change, the predicted 4.5 million Australians diagnosed with COPD by 2050 will place significant burdens on already over-utilised frontline ED services. Comment The calculation reveals a normal gradient, indicating that the etiology for hypoxemia and hypoventilation is extrinsic to the lung itself. Many in this series of 12- to 20 . This one-page fact sheet focuses on preventing pneumonia in individuals with COPD. overview of copd recap basic knowledge update, COPD - . Support groups. COPD - . Views: 991, By: sherry By whitelisting SlideShare on your ad-blocker, you are supporting our community of content creators. what is, COPD - . Free access to premium services like Tuneln, Mubi and more. COPD 1 / 32. Presentations will focus on current and critical issues to provide oncology nurses with practical information that can be implemented in the practice setting. causes. Normally, the airways and air sacs in your lungs are elastic or stretchy. Comprises primarily of two related disease- Lung cancer Clubbing of the digits is not a sign of COLD.In patients with COLD, development of lung cancer is the most likely explanation for newly developed clubbing. Pharmacotherapy of Chronic Obstructive Pulmonary Disease, CODP ( Chronic Obstructive Pulmonary Disease ). About the Position: As the Area Practice Manager, you will oversee the operations of all Pulmonary and Critical Care Services (Critical Care providers only), including overseeing 40+ colleagues including: 11 physicians, 9 providers and APPs, 10 MAs/RNs, 1 Nursing Supervisor, 1 Assistant Clinical Lead, Lung Nodule Nurse Navigator, LN Program . -Continuous evaluation of classes and the course to . This n urse PowerPoint template for free comes with many medical illustrations. Avg rating:3.0/5.0. Study Resources. Please Review ONE of the following videos and post for the class what you have learned. c hronic o bstructive, COPD - . or chronic bronchitis that was dr bruce davies. Clipping is a handy way to collect important slides you want to go back to later. Bandaged Teddy Bear - Nursing Templates Free. Enjoy access to millions of ebooks, audiobooks, magazines, and more from Scribd. COPD PowerPoint Presentation. American Lung Association; Better Breathers Club. Activate your 30 day free trialto unlock unlimited reading. Tap here to review the details. Updated November 2021. Blood gases drawn on room air revealed these values: patient 1- pH =7.18, PCO2 = 70mmHg, PO2=50mmHg, HCO3=24mEq/L; patient2- pH =7.31, PCO2=50mmHg, PO2=50mmHg, HCO3=25mEq/L, Comment The A-a gradient calculation for patient 1 is as follows: A-a DO2 = PAO2 PaO2 PAO2 = 150 (1.25x PCO2) PAO2 = 150 (1.25x 70) PAO2 = 62 A-a =62 50 A-a = 12. Tachypnea Accessory respiratory muscle use. joshua jewell. A chest X-ray can show emphysema, one of the main causes of COPD. 28-7, Emphysema Pathophysiology Hyperinflation of alveoli Destruction of alveolar walls Destruction of alveolar capillary walls Narrowed airways Loss of lung elasticity, Emphysema Pathophysiology Two types: Centrilobular (central part of lobule) Most common Panlobular (destruction of whole lobule) Usually associated with AAT deficiency, Emphysema Pathophysiology Structural changes are: Hyperinflation of alveoli Destruction of alveolar capillary walls Narrowed, tortuous small airways Loss of lung elasticity, Emphysema Pathophysiology Small bronchioles become obstructed as a result of Mucus Smooth muscle spasm Inflammatory process Collapse of bronchiolar walls Recurrent infections production/stimulation of neutrophils and macrophages release proteolytic enzymes alveolar destruction inflammation, exudate, and edema, Emphysema Pathophysiology Elastin and collagen are destroyed Air goes into the lungs but is unable to come out on its own and remains in the lung Causes bronchioles to collapse, Emphysema Pathophysiology Trapped air hyperinflation and overdistention As more alveoli coalesce, blebs and bullae may develop Destruction of alveolar walls and capillaries reduced surface area for O2 diffusion Compensation is done by increasing respiratory rate to increase alveolar ventilation Hypoxemia usually develops late in disease, Emphysema Clinical Manifestations Dyspnea Progresses in severity Patient will first complain of dyspnea on exertion and progress to interfering with ADLs and rest, Emphysema Clinical Manifestations Minimal coughing with no to small amounts of sputum Overdistention of alveoli causes diaphragm to flatten and AP diameter to increase, Emphysema Clinical Manifestations Patient becomes chest breather, relying on accessory muscles Ribs become fixed in inspiratory position, Emphysema Clinical Manifestations Patient is underweight (despite adequate calorie intake), Chronic Bronchitis Pathophysiology Pathologic lung changes are: Hyperplasia of mucus-secreting glands in trachea and bronchi Increase in goblet cells Disappearance of cilia Chronic inflammatory changes and narrrowing of small airways Altered fxn of alveolar macrophages infections, Chronic Bronchitis Pathophysiology Chronic inflammation Primary pathologic mechanism causing changes Narrow airway lumen and reduced airflow d/t hyperplasia of mucus glands Inflammatory swelling Excess, thick mucus, Chronic Bronchitis Pathophysiology Greater resistance to airflow increases work of breathing Hypoxemia and hypercapnia develop more frequently in chronic bronchitis than emphysema, Chronic Bronchitis Pathophysiology Bronchioles are clogged with mucus and pose a physical barrier to ventilation Hypoxemia and hypercapnia d/t lack of ventilation and O2 diffusion Tendency to hypoventilate and retain CO2 Frequently patients require O2 both at rest and during exercise, Chronic Bronchitis Pathophysiology Cough is often ineffective to remove secretions because the person cannot breathe deeply enough to cause air flow distal to the secretions Bronchospasm frequently develops More common with history of smoking or asthma, Chronic Bronchitis Clinical Manifestations Earliest symptoms: Frequent, productive cough during winter Frequent respiratory infections, Chronic Bronchitis Clinical Manifestations Bronchospasm at end of paroxysms of coughing Cough Dyspnea on exertion History of smoking Normal weight or heavyset Ruddy (bluish-red) appearance d/t polycythemia (increased Hgb d/t chronic hypoxemia)) cyanosis, Chronic Bronchitis Clinical Manifestations Hypoxemia and hypercapnia Results from hypoventilation and airway resistance + problems with alveolar gas exchange, COPD Complications Pulmonary hypertension (pulmonary vessel constriction d/t alveolar hypoxia & acidosis) Cor pulmonale (Rt heart hypertrophy + RV failure) Pneumonia Acute Respiratory Failure, COPD Diagnostic Studies Chest x-rays early in the disease may not show abnormalities History and physical exam Pulmonary function studies reduced FEV1/FVC and residual volume and total lung capacity, COPD Diagnostic Studies ABGs PaO2 PaCO2 (especially in chronic bronchitis) pH (especially in chronic bronchitis) Bicarbonate level found in late stages COPD, COPD Collaborative Care Smoking cessation Most significant factor in slowing the progression of the disease, COPD Collaborative Care: Drug Therapy Bronchodilators as maintenance therapy -adrenergic agonists (e.g. COPD Chronic obstructive pulmonary disease (COPD) is a lung ailment that is characterized by a persistent blockage of airflow from the lungs. 1.) Chronic obstruction of the flow of air through . all you wanted to know about copd but were afraid to ask. If you would like to sign up for our eNewsletter please click the button below. An X-ray can also rule out other lung problems or heart failure. Special considerations in design of clinical trials for special Disease condi Polices for intensive care units / critical care units, RESUSCIATION EQUIPMENTS IN INTENSISIVE CARE UNITS, Nursing management of critically ill patient in intensive care units, Infection control protocols in intensive care units, Nursing management of patients with oncological conditions, Nursing assessment and management of patients with hepatic disorders, Nursing assessment and Management clients with Pancreatic disorders, V Bobic - Mucoid ACL - ACL SG St Kitts 2023 As presented.pdf, Unit-1 New drug discovery and drug development PPT.ppt, Checklist for lab and cleanroom environ mental monitoring systems, UNIT 4 TOPIC Managing Monitoring Clinical Trials.pdf. The COPD Pocket Consultant Guide (PCG) app is built to support patients with COPD and their family members in self-management and to assist health care professionals in providing optimal care. COPD - Physical Findings. mary care nurses can be pivotal in the early identification, assessment and manage-ment of COPD (Hooper et al, 2012). 26 slides. chronic obstructive pulmonary disease. Avoid or control exposure to occupational and environmental pollutants and irritants Early detection of small-airway disease Early diagnosis of respiratory tract infections, Nursing Management Nursing Implementation Acute Intervention Required for complications like pneumonia, cor pulmonale, and acute respiratory failure, Nursing Management Nursing Implementation Ambulatory and Home Care Pulmonary rehabilitation Control and alleviate symptoms of pathophysiologic complications of respiratory impairment, Nursing Management Nursing Implementation Ambulatory and Home Care Teach patient how to achieve optimal capability in carrying out ADLs Physical therapy Nutrition Education Activity considerations Exercise training of upper extremities to help improve function and relieve dyspnea, Nursing Management Nursing Implementation Ambulatory and Home Care Explore alternative methods of ADLs Encourage patient to sit while performing activities Coordinated walking, Nursing Management Nursing Implementation Ambulatory and Home Care Slow, pursed-lip breathing After exercise, wait 5 minutes before using -adrenergic agonist MDI, Nursing Management Nursing Implementation Ambulatory and Home Care Sexual activity Plan during part of day when breathing is best Slow, pursed-lip breathing Refrain after eating or other strenuous activity Do not assume dominant position Do not prolong foreplay, Nursing Management Nursing Implementation Ambulatory and Home Care Sleep Nasal saline sprays Decongestants Nasal steroid inhalers Long-acting theophylline Decreases bronchospasm and airway obstruction, Nursing Management Nursing Implementation Ambulatory and Home Care Psychosocial considerations Guilt Depression Anxiety Social isolation Denial Dependence Use relaxation techniques and support groups. Views: 670, By: DrDwayne . chronic obstructive pulmonary disease. In the United States alone it affects about 16 million people. In addition, laminated versions are available through our online catalogue. Weakness. by Anna Curran. Pneumothorax, or a collapsed lung, is the collection of air in the spaces around the lungs. Chronic Obstructive California University Balance Between Formal Teams Communities of Practice Re California State University Light a Candle or Curse the Darkness.docx, California State University Utilitarianism Kantian Ethics Videos Questions.docx. having lower oxygen levels than normal . This is an original nursing presentation for PowerPoint. Activate your 30 day free trialto continue reading. Views: 837, By: DrDwayne Appointments 216.444.6503. COPD - . By whitelisting SlideShare on your ad-blocker, you are supporting our community of content creators. pulmonary diseases. The disease affects millions of Americans and is a leading cause of disability and death in the U.S. courtney pearce & jenna paterno . As pulmonary sequelae constitute a serious long COVID subtype, exploring patient experience and needs can generate knowledge to guide nursing practice. California State Musculoskeletal Metabolic and Multisystem Health Dysfunction Callimbah is a successful company that is involved in the.docx, California State University Economists and Finance of Tesco Presentation.docx. Causes and types of chronic obstructive . Activate your 30 day free trialto unlock unlimited reading. Chronic Obstructive Pulmonary Disease (COPD) Slide 2-. Chronic obstructive pulmonary disease (COPD)is a disease state characterized by airflow limitation that is not fully reversible. By accepting, you agree to the updated privacy policy. Chronic obstructive pulmonary disease, also known as COPD, encompasses a group of diseases that cause problems with breathing. The SlideShare family just got bigger. Log in Join. management of stable copd shyam rao may 2014. objectives . This booklet covers many important topics, such as how bronchiectasis is diagnosed, treatment options, tips for living well and reducing exacerbations, and how to find support for living with bronchiectasis. Physiotherapy management of chronic obstructive pulmonary disease ppt by Oluw COPD AND ICU MANAGEMENT : DR DEVAWRAT BUCHE, treatment Chronic Obstructive Pulmonary Disease. advance their professional expertise with presentations and earn more on top of their base rate.-High Reliability Organization (HRO): MedStar is an HRO. Learn faster and smarter from top experts, Download to take your learnings offline and on the go. The presentation must educate advanced practice nurses on assessment and care/treatment, including . Tap here to review the details. Contact Hours: 42.5 This nursing continuing professional development activity was approved by the American Association of Critical-Care Nurses, an accredited approver by the American Nurses Credentialing Center's Commission on Accreditation. working around certain kinds of chemicals, COPD - . Systemic wasting Significant weight loss Bitemporal wasting Diffuse loss of subcutaneous adipose tissue Paradoxical respiration Inward movement of the rib cage with inspiration (Hoover's sign) in some patients "Pink puffers" are patients with predominant emphysemano cyanosis or edema, with decreased breath sounds. You can read more about the PCG tracks for patients and caregivers and for health professionals. Faster and smarter from top experts, Download to take your learnings offline and the! More about the PCG tracks for patients and caregivers and for health care professionals practice nurses on assessment and of! 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